1. Introduction & Purpose
Since the publication of “Science Digest” March 1978 issue reporting colloidal
Silver in the article “Our Mightiest Germ Fighter” silver is emerging as a wonder
of modern medicine. Science has traveled a long way in recognizing the fact that
an antibiotic kills perhaps a half-dozen different disease organism , but silver kills
some 650. Also resistent strains fail to develop against silver. Moreover, silver is
virtually nontoxic to humans,
Obviously the question that would came to mind
is how to deliver such colloidal silver internally. Ssophagus BE -. Stomoach
pm i
A standard dose of 10 ppm to 40 ppm aqueous
colloidal silver is normally two spoons (~ 10ml)
at a time. This brings up the question as to the
fate of colloidal silver in the digestive systems as
it travels through various zones of digestive :
system. It may undergo convertion in the acidic Ji LEAL intestine
condition of the stomach. It is absorbed in the E
duodenum. The remaining silver as it travels Rectum
further inevitably contacts intestinal flora.
All of us carry in our intestinal tracts a complex ecosystem of microbes. These
bacteria are highly important to our health, providing us with protection against
intestinal infection, supplying us with additional nutritional value from the food we
eat, and contributing to the development of our immune system. In fact
repopulation of the gut microflora, after or during antibiotic therapy is
accomplished thru OTC or prescription lactobacilli formulations. The flip-side is
that disturbances in this ecosystem can leave us more vulnerable to exogenous and
endogenous intestinal infections.
Lacto bacilli taken as a probiotic supplementation, may also concurrently be
travelling with colloidal silver through the digestive tract. The concept of
ingesting live organism for the purpose of improving one’s intestinal health and
general well being is around as old as curds (yogurt) to capsules of Lacto bacilli.
Question L. : Does this “Top Gun’ (colloidal silver), that destroys more than 650
microorganisms, spares the “good guys” – intestinal Lacto bacilli
flora.?
Question 2. : And if indeed it spares Lacto bacilli then the question arises how
does it distinguish between good and the toxic.?
Question 3. : Isn’t the mechanism to spare the good bacteria applicable to any
other pathogenic bacteria?
This report answers the first question while study is well into answering
Questions 2 and 3.
2. Aim
To confirm the hypothesis that ASAP silver solutions are not toxic to
Probiotics such as
1. Lactobacillus & Bifidobacterium supplementss.
2. Multistrain Oral Preparations
a. Lactisyn :Mfg. By Laboratories Griffon Pvt. Ltd. India
Composition :
Lactobacillus lactis
Lactobacillus acidophilus
Streptococcus lactis
Streptococcus thermophilus
b. Kyo-Dophilus :Mfg. By Wakunaga Of America Co. Ltd., USA
Marketed by : GNC, USA.
Composition :
Lactobacillus acidophilus
Bifidobacterium bifidum
Bifidobacterium longum
Table — 1 Effect of ASAP on Lactic acid Bacteria
. Diameter of zone of Inhibition (mm)
Name of Medium ASAP 14ppm Photog
Product Used ASAP 10ppm +H,0, ASAP 22ppm raphs
Lactobacillus GYEA NI 36 NI
acidophillus TA NI 30 NI 1
Bifidobacterium GYEA NI 35 NI 2
longum TA NI 28 NI
. GYEA Ni 46 NI
Lactisyn TA NI 36 NI 3
y , GYEA NI 48 NI
Kyo-Dophilus TA NI 35 NM 4
Key NI : No Inhibition
Table — 1 Effect of ASAP on Lactic acid Bacteria
Name of Medium De or | ites (mm) Fotos
Product Used ASAP 10ppm +H,0; ASAP 22ppm raphs
Lactobacillus GYEA NI 16 NI
acidophillus TA NI 30 NI 1
Bifidobacterium GYEA NI 35 NI 2
longum TA MN 28 Ni
Lactisyn TA MN 36 NI 3
, . GYEA NI 48 NI
Kyo-Dophilus TA NI 3 NI 4
Key NI : No Inhibition
7. Conclusion
ASAP solutions at both 10 & 22 ppm concentrations have
not demonstrated anti-probiotic activity. ASAP 14 ppm with 1.5% H,0,
shows appreciable killing of all the strains tested presumably due to H,;0,.
The results bring forth ASAP as an “antibiotic of choice”, natural
antibiotic, with firstly no side effects such as diarrhea and infact will not
disturb the body’s natural host defense mechanism. It indeed complements
therapy by sparing essential host microflora as well as concomittant oral
lactobacilli therapy normally given as an adjunct.